By Ambrosetti A., Malchiodi A.
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Additional info for A multiplicity result for the Yamabe problem on S n
Binding proteins dissociate upon ligand binding Receptor-Binding Proteins APC in GI Cancers ! FAP due to germline mutations in APC ! Somatic (sporadic) mutations in the APC gene initiate colorectal malignancy ! APC mutations in 63% of colonic adenomas ! 99% of germline and 100% of sporadic APC mutations lead to truncated protein Significance of Signal Transduction to Disease: Tumorigenesis ● ● Cancer cells contain genetic damage many of the genes encoding signaling molecules have been shown to be capable of causing cancer if disrupted in some way APC Function !
ABL gene on chromosome 9 placed next to BRC gene on 22 results in a fusion protein bcr-abl ! abl is a non-receptor Y-kinase ! bcr is member of the Rho family of small GTPases ! Similar translocation occurs in ALL CML is target of Y-kinase inhibitor Glivec Non-Hodgkin’s Lymphoma ! ! !
APC mutations in 63% of colonic adenomas ! 99% of germline and 100% of sporadic APC mutations lead to truncated protein Significance of Signal Transduction to Disease: Tumorigenesis ● ● Cancer cells contain genetic damage many of the genes encoding signaling molecules have been shown to be capable of causing cancer if disrupted in some way APC Function ! ! APC associates with β-catenin, a protein involved in the Wnt signaling pathway APC induces β-catenin phos. g. E-cadherin Wnt signaling β-catenin bound in complex which includes GSK-3; this kinase phosphorylates βcatenin leading to its degradation.
A multiplicity result for the Yamabe problem on S n by Ambrosetti A., Malchiodi A.